指导或协助指导博士研究生11人，硕士研究生5人，目前已毕业6人，在读10人。已毕业研究生均在妇科肿瘤精准医学的基础与临床研究领域有较好的科研产出或已申获国家自然科学基金青年项目。另外，作为检验科副主任主管科室科研工作，带领检验科科研团队32人，在细胞遗传、临检、微生物、生化、免疫等多个检验亚学科领域开展研究工作。

国家自然科学基金面上项目（批号：82272992），mTORC1-IBTK-eIF4A1信号轴通过上调翻译起始促进宫颈癌的分子机制，2023.1-2026.12
上海市自然科学基金面上项目（批号：22ZR1449200），组蛋白甲基转移酶GLP /G9a通过上调PD-L1表达促进内膜癌细胞免疫逃逸的分子机制研究，2022.4-2025.3
国家自然科学基金重大研究计划培育项目（批号：91954106），能量压力下INF2蛋白翻译后修饰动态调控内质网-线粒体互作的分子机制及其在内膜癌发生中的作用研究，2020.1-2022.12
国家自然科学基金面上项目（批号：81872109），子宫内膜癌SPOP基因突变促进PD-L1介导的肿瘤免疫逃逸的机制研究，2019.1-2022.12
上海市自然科学基金面上项目（批号：18ZR1430100），子宫内膜癌SPOP基因突变在肿瘤免疫逃逸中的作用机制研究，2018.5-2021.4

1. 近三年以第一或通讯作者发表论文情况：
2. Chen Y, Jiao D, He H, Sun H, Liu Y, Shi Q, Zhang P, Li Y, Mo R*, Gao K*, Wang C*. Disruption of the Keap1-mTORC2 axis by cancer-derived Keap1/mLST8 mutations leads to oncogenic mTORC2-AKT activation. Redox Biol. 2023 Sep 5;67:102872. 
3. Gao K*, Shi Q, Gu Y, Yang W, He Y, Lv Z, Ding Y, Cao W, Wang C*, Wan X*. SPOP mutations promote tumor immune escape in endometrial cancer via the IRF1-PD-L1 axis. Cell Death Differ. 2023 Feb;30(2):475-487. 
4. Wang M, Yan Q, Song Y, Zhang Z, Chen X, Gao K*, Wan X*. Loss-of-function mutations of SOX17 lead to YAP/TEAD activation-dependent malignant transformation in endometrial cancer. Oncogene. 2023 Jan;42(4):322-334. 
5. Lin Q, Chen Z, Shi W, Lv Z, Wan X*, Gao K*. JAK1 inactivation promotes proliferation and migration of endometrial cancer cells via upregulating the hypoxia-inducible factor signaling pathway. Cell Commun Signal. 2022 Nov 14;20(1):177.
6. Gao K*, Shi Q, Liu Y, Wang C*. Enhanced autophagy and NFE2L2/NRF2 pathway activation in SPOP mutation-driven prostate cancer. Autophagy. 2022 Aug;18(8):2013-2015. 
7. Lv Z, Ding Y, Cao W, Wang S*, Gao K*. Role of RHO family interacting cell polarization regulators (RIPORs) in health and disease: Recent advances and prospects. Int J Biol Sci. 2022 Jan 1;18(2):800-808. 
8. Shi Q, Jin X, Zhang P, Li Q, Lv Z, Ding Y, He H, Wang Y, He Y, Zhao X, Zhao SM, Li Y, Gao K*, Wang C*. SPOP mutations promote p62/SQSTM1-dependent autophagy and Nrf2 activation in prostate cancer. Cell Death Differ. 2022 Jun;29(6):1228-1239. 
9. Zhang P*, Gao K*, Zhang L, Sun H, Zhao X, Liu Y, Lv Z, Shi Q, Chen Y, Jiao D, Li Y, Gu W*, Wang C*. CRL2-KLHDC3 E3 ubiquitin ligase complex suppresses ferroptosis through promoting p14^ARF degradation. Cell Death Differ. 2022 Apr;29(4):758-771.
10. Zhang J#, Gao K#, Xie H, Wang D, Zhang P, Wei T, Yan Y, Pan Y, Ye W, Chen H, Shi Q, Li Y, Zhao SM, Hou X, Weroha SJ, Wang Y, Zhang J, Karnes RJ, He HH, Wang L, Wang C*, Huang H*. SPOP mutation induces DNA methylation via stabilizing GLP/G9a. Nat Commun. 2021 Sep 29;12(1):5716. 
11. Jin X*, Qing S, Li Q, Zhuang H, Shen L, Li J, Qi H, Lin T, Lin Z, Wang J, Cao X, Yang J, Ma Q, Cong L, Xi Y, Fang S, Meng X, Gong Z, Ye M, Wang S*, Wang C*, Gao K*. Prostate cancer-associated SPOP mutations lead to genomic instability through disruption of the SPOP-HIPK2 axis. Nucleic Acids Res. 2021 Jul 9;49(12):6788-6803.
12. Wen X, Wan J, He Q, Wang M, Li S, Jiang M, Qian Z, Liu B, Lu W, Wang K, Gao K*, Wan X*. p190A inactivating mutations cause aberrant RhoA activation and promote malignant transformation via the Hippo-YAP pathway in endometrial cancer. Signal Transduct Target Ther. 2020 May 27;5(1):81.